00:01
So we want to talk now about secondary
lesions.
00:03
We've finished discussing primary lesions.
00:06
We're moving on to secondary lesions a crust
it is a dried serum can be serous blood or
pus on the surface of the skin.
00:17
It may include bacteria typically
Staphylococcus aureus which we find on the
skin. Here are some examples of crusts.
00:26
This is a patient with impetigo secondarily
infected with staph aureus bacteria,
resulting in crusting which we have just
described.
00:36
The color may be honey colored or it may be
hemorrhagic.
00:41
The next topic is about the next type is
scale.
00:46
In a scale, what do we see?
We see hyperkeratosis that is thickening of
the skin, and this is due to accumulation of
stratum corneum due to increased
proliferation and or delayed desquamation.
00:59
These are some examples of scale.
01:02
This is a patient with typical psoriasis
vulgaris with silvery scales on the buttock
area. Another type of scale is a patient
with tinea corporis.
01:14
You can see an active annular scale on this
picture.
01:20
A fissure.
01:21
This is a linear cleft in skin, and it's
usually painful.
01:26
It results from marked drying, skin
thickening, and loss of elasticity.
01:31
An example of a fissure is angular colitis.
01:35
We see angular colitis in patients who have
either diabetes or immunocompromised from
HIV. And sometimes it can be used to it can
be used to ill feature fitting dentures.
01:50
Excoriation is an exogenous injury to all or
part of the dermis.
01:55
It is usually due to scratching.
01:57
An example of an excoriation.
01:59
This is a young girl who's been picking the
skin, resulting in excoriations.
02:05
You can also see excoriations in patients
with atopic eczema who have intense pruritis,
and they tend to pick and scratch on the
skin.
02:14
The next type of secondary lesion is
lichenification, which is due to a we call it
acanthosis, which simply means thickening of
the epidermis and accentuation of natural
skin lines. So these tend to be the skin
lines tend to be more obvious, and this
results from repeated scratching or rubbing.
02:35
This is an example of a patient with a
thickening of the skin or lichenification,
because it's due to lichen planus, lichen
planus is itchy, and patients who have like
lichenification, it's usually due to itchy
conditions.
02:53
The example here is that of a
lichenification due You to chronic atopic
dermatitis. As I mentioned, you tend to see
lichenification in patients who have got
intense pruritis of each.
03:07
An erosion is partial or sometimes complete
loss of the epidermis, and it's moist, oozing
and or crusted lesion.
03:16
This is a young patient with a
staphylococcal scalded skin syndrome SS,
where you see typical erosions due to the
lifting of the epidermis.
03:27
You can actually see typical erosions in
patients with pemphigus vulgaris when the
blister is actually opened.
03:35
Ulceration involves deeper defect compared
to an erosion loss of the entire epidermis
and the superficial dermis.
03:45
The examples of accelerated ulceration are
scrofula, edema, which is a type of cutaneous
tuberculosis, and this is due to infection
with the TB bacteria.
03:56
Another example of an ulcer is diabetic foot
ulcer.
04:01
Moving on now to a atrophy.
04:05
This can be epidermal dermal or a combination
of these two.
04:10
If it's epidermal, it means there's thinning
of the epidermis, leading to wrinkling and
shiny appearance.
04:16
When it's dermal, you get loss of the
dermis, particularly the collagen and
elastin, leading to a depression on the
skin.
04:25
An example of this is, for example, patients
with senile atrophy, where the skin is so
thin because of the loss of collagen and
photoaging.
04:35
Another example is anetoderma, where you
also see wrinkling of the skin, almost like
tissue paper.
04:42
What about scars?
We all complain about scars when you've got
acne or after injury, so this is an important
aspect of dermatology.
04:50
So what do we mean by scar.
04:52
It's increased thickness of the damage
caused by enhanced production of collagen by
the fibroblasts. And remember we mentioned
that we tend to get more fibroblasts in black
skin or melanin rich skin than in light
skin.
05:05
So automatically we do anticipate that we
will have more keloids and scars in black
skin. So this is an example of a scar.
05:16
How do you differentiate between a scar and
a keloid?
Scar is limited to the original wound
margin.
05:24
That's what we call a hypertrophic scar.
05:27
And it can be painful.
05:29
And the keloid is hypertrophic scar that
extends beyond the original wound margin.
05:37
And that's how you differentiate between the
two.
05:39
And by the way, you can also get spontaneous
keloid where there is no history of any
wound. But patients get spontaneous keloid.
05:48
And we see this a lot amongst patients of
skin of color.